G Protein Signaling Pathways in Health and Disease

G Protein Signaling Pathways in Health and Disease

Tao, Ya-Xiong

126,88 €(IVA inc.)

G Protein Signaling Pathways in Health and Disease, Volume 161 in the Progress in Molecular Biology and Translational Science series, provides informative and exciting monographs on a wide variety of research topics related to G Protein Signaling Pathways in Health and Disease. The series gives in-depth knowledge on the important molecular biological aspects of organismal physiology and function, along with insights on how this knowledge can be applied to understand and ameliorate human disease. This updated release covers Diseases associated with mutations in CNGA3, Mutations in arrestins, Diseases caused by mutations in GPR101, Diseases caused by mutations in lutropin receptor, and much more. Comprises 15-20 chapters, providing substantial coverage on a given topicContains ample use of tables, diagrams, schemata and color figures to enhance the reader's ability to rapidly grasp the information provided in each chapterProvides a comprehensive guide to the latest information available on prions, viruses, bacteria, and eukaryotes INDICE: 1. Introduction to G protein signaling pathways Ya-Xiong Tao 2. Diseases associated with mutations in CNGA3 Qingjiong Zhang 3. Mutations in arrestins Vsevolod V. Gurevich 4. Diseases caused by mutations in GPR101 Ya-Xiong Tao 5. Diseases caused by mutations in lutropin receptor Jie Qiao 6. Beta3-adrenergic receptor polymorphism and type 2 diabetes Ya-Xiong Tao 7. Chemokine receptor mutations and susceptibility to HIV infection Yi-Qun Kuang 8. GPR154 mutations and asthma Ya-Xiong Tao 9. Diseases associated with mutations in prokineticin receptor-2 gene Jia-Da Li 10. Therapeutic options for loss-of-function mutations in GPCRs Wei Wang 11. GPCRs and primary cilia Ya-Xiong Tao

  • ISBN: 978-0-12-816724-3
  • Editorial: Academic Press
  • Encuadernacion: Cartoné
  • Páginas: 300
  • Fecha Publicación: 01/02/2019
  • Nº Volúmenes: 1
  • Idioma: Inglés